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Asish K. Ghosh, PhD
Pathology

Asish K. Ghosh, PhD

Professor
Section of Experimental Pathology


The University of Oklahoma Health Sciences Center
Stephenson Cancer Center
975 NE 10th St, BRC 417B
Oklahoma City, OK 73104

405-271-1387

asish-ghosh@ouhsc.edu


Education:

  • MS - Biochemistry, University of Calcutta, India (1987)
  • PhD - Biochemistry, University of Calcutta, India (1998)


Fellowship:

  • Research Fellow -  Department of Microbiology, Bose Institute, Calcutta, India (1989-1997)
  • Post-Doctoral Fellow - Department of Pathology, St. Louis University, St. Louis, MO (1998-1999)
  • Research Associate - Department of Pathology, St. Louis University, St. Louis, MO (1999-2001)
  • Research Associate - Division of Hematology, Mayo Clinic, Rochester, MN (2006-2008)


Clinical/Research Interests:

  • Chronic Lymphocytic Leukemia (CLL) cell biology
  • Tyrosine Kinase (RTK & non-RTK) signaling
  • Cell Survival Signaling Pathways
  • Drug Targeting
  • Small Molecule Inhibitors
  • B Cell Lymphomas
  • Tumor Microenvironment/Extracellular
  • Vesicles


Funding:

R21CA274072      Asish Ghosh (PI)        7/7/2022 – 6/30/2025 (NCE)
NIH/NCI
Study Pro-oncogenic Role of BCL6 in CLL Tumorigenesis.
The proposed studies have potential to establish a new prognostic parameter and therapeutic avenue via targeting BCL6 in CLL cells in combination with the current BCR-targeted therapy, ibrutinib. Tumorigenesis.
Role: PI

R21CA274072      Asish Ghosh (PI)         4/10/2023 – 3/31/2025
NIH/NCI
Pro-Oncogenic Role of Mitochondrial Lipid Kinase in CLL.
The proposed studies have enormous potential to establish a new prognostic parameter and therapeutic avenue via targeting AGK in CLL cells with or without the current BCR-targeted therapy, ibrutinib.
Role: PI


Select Honors and Accomplishments:

  • Fellowships from the Council of Scientific and Industrial Research (CSIR), Government of India
  • Young Investigator Award, Mayo Clinic Angiogenesis Symposium (2008)
  • Hematology Research Summit Award Recipient, Mayo Clinic (2012)


Select Publications:

  1. Mamidi MK, et al. Idelalisib activates AKT via increased recruitment of PI3Kdelta/PI3Kbeta to BCR signalosome while reducing PDK1 in post-therapy CLL cells. Leukemia. 2022. Epub 20220514. doi: 10.1038/s41375-022-01595-0. PubMed PMID: 35568768.
  2. Maiti GP, et al. SIRT3 overexpression and epigenetic silencing of catalase regulate ROS accumulation in CLL cells activating AXL signaling axis. Blood Cancer J. 2021;11(5):93. Epub 2021/05/19. doi: 10.1038/s41408-021-00484-6. PubMed PMID: 34001853; PMCID: PMC8129117.
  3. Sinha S, et al. Targeted Axl Inhibition Primes Chronic Lymphocytic Leukemia B Cells to Apoptosis and Shows Synergistic/Additive Effects in Combination with BTK Inhibitors. Clinical cancer research : an official journal of the American Association for Cancer Research. 2015;21(9):2115-26. Epub 2015/02/13. doi: 10.1158/1078-0432.CCR-14-1892. PubMed PMID: 25673699.